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g6pd reagent set pointe scientific g7583180  (Pointe Scientific)

 
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    Pointe Scientific g6pd reagent set pointe scientific g7583180
    Characterization of <t>G6PD</t> Med− mice. (A) mQTL analysis of samples from 13 091 blood donors highlights correlation of succinate levels with polymorphic G6PD. (B) Locus zoom identifying G6PD as a target of interest. (C) An overview of glucose metabolism in redox homeostasis in RBCs. (D) Total abundance of 6-phosphogluconate (6P-gluconate) from metabolic tracing (sum of labeled and unlabeled) present in genotypes. (E) G6PD activity assay; (F) PPP activation as judged by relative levels of labeled 6P-gluconate to hexose phosphate. (G-H) CS testing of mice (n = 12) showed that G6PD Med− mice maintained a significantly faster CS (8% increase). Dashed lines indicate CS. (I) G6PD Med− mice have higher anaerobic work capacity (AWC) as measured by the slope, hG6PD ND = 1715 and hG6PD MED- = 945.4 (significance, ∗ P < .05, ∗∗ P < .01, ∗∗∗∗ P < .0001).
    G6pd Reagent Set Pointe Scientific G7583180, supplied by Pointe Scientific, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/set%2C+pointe+scientific/pmc11786683-31-6-9?v=Pointe+Scientific
    Average 90 stars, based on 1 article reviews
    g6pd reagent set pointe scientific g7583180 - by Bioz Stars, 2026-06
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    1) Product Images from "Increased exercise tolerance in humanized G6PD-deficient mice"

    Article Title: Increased exercise tolerance in humanized G6PD-deficient mice

    Journal: Blood Advances

    doi: 10.1182/bloodadvances.2024013968

    Characterization of G6PD Med− mice. (A) mQTL analysis of samples from 13 091 blood donors highlights correlation of succinate levels with polymorphic G6PD. (B) Locus zoom identifying G6PD as a target of interest. (C) An overview of glucose metabolism in redox homeostasis in RBCs. (D) Total abundance of 6-phosphogluconate (6P-gluconate) from metabolic tracing (sum of labeled and unlabeled) present in genotypes. (E) G6PD activity assay; (F) PPP activation as judged by relative levels of labeled 6P-gluconate to hexose phosphate. (G-H) CS testing of mice (n = 12) showed that G6PD Med− mice maintained a significantly faster CS (8% increase). Dashed lines indicate CS. (I) G6PD Med− mice have higher anaerobic work capacity (AWC) as measured by the slope, hG6PD ND = 1715 and hG6PD MED- = 945.4 (significance, ∗ P < .05, ∗∗ P < .01, ∗∗∗∗ P < .0001).
    Figure Legend Snippet: Characterization of G6PD Med− mice. (A) mQTL analysis of samples from 13 091 blood donors highlights correlation of succinate levels with polymorphic G6PD. (B) Locus zoom identifying G6PD as a target of interest. (C) An overview of glucose metabolism in redox homeostasis in RBCs. (D) Total abundance of 6-phosphogluconate (6P-gluconate) from metabolic tracing (sum of labeled and unlabeled) present in genotypes. (E) G6PD activity assay; (F) PPP activation as judged by relative levels of labeled 6P-gluconate to hexose phosphate. (G-H) CS testing of mice (n = 12) showed that G6PD Med− mice maintained a significantly faster CS (8% increase). Dashed lines indicate CS. (I) G6PD Med− mice have higher anaerobic work capacity (AWC) as measured by the slope, hG6PD ND = 1715 and hG6PD MED- = 945.4 (significance, ∗ P < .05, ∗∗ P < .01, ∗∗∗∗ P < .0001).

    Techniques Used: Labeling, Activity Assay, Activation Assay



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    Characterization of <t>G6PD</t> Med− mice. (A) mQTL analysis of samples from 13 091 blood donors highlights correlation of succinate levels with polymorphic G6PD. (B) Locus zoom identifying G6PD as a target of interest. (C) An overview of glucose metabolism in redox homeostasis in RBCs. (D) Total abundance of 6-phosphogluconate (6P-gluconate) from metabolic tracing (sum of labeled and unlabeled) present in genotypes. (E) G6PD activity assay; (F) PPP activation as judged by relative levels of labeled 6P-gluconate to hexose phosphate. (G-H) CS testing of mice (n = 12) showed that G6PD Med− mice maintained a significantly faster CS (8% increase). Dashed lines indicate CS. (I) G6PD Med− mice have higher anaerobic work capacity (AWC) as measured by the slope, hG6PD ND = 1715 and hG6PD MED- = 945.4 (significance, ∗ P < .05, ∗∗ P < .01, ∗∗∗∗ P < .0001).
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    Characterization of <t>G6PD</t> Med− mice. (A) mQTL analysis of samples from 13 091 blood donors highlights correlation of succinate levels with polymorphic G6PD. (B) Locus zoom identifying G6PD as a target of interest. (C) An overview of glucose metabolism in redox homeostasis in RBCs. (D) Total abundance of 6-phosphogluconate (6P-gluconate) from metabolic tracing (sum of labeled and unlabeled) present in genotypes. (E) G6PD activity assay; (F) PPP activation as judged by relative levels of labeled 6P-gluconate to hexose phosphate. (G-H) CS testing of mice (n = 12) showed that G6PD Med− mice maintained a significantly faster CS (8% increase). Dashed lines indicate CS. (I) G6PD Med− mice have higher anaerobic work capacity (AWC) as measured by the slope, hG6PD ND = 1715 and hG6PD MED- = 945.4 (significance, ∗ P < .05, ∗∗ P < .01, ∗∗∗∗ P < .0001).
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    Characterization of G6PD Med− mice. (A) mQTL analysis of samples from 13 091 blood donors highlights correlation of succinate levels with polymorphic G6PD. (B) Locus zoom identifying G6PD as a target of interest. (C) An overview of glucose metabolism in redox homeostasis in RBCs. (D) Total abundance of 6-phosphogluconate (6P-gluconate) from metabolic tracing (sum of labeled and unlabeled) present in genotypes. (E) G6PD activity assay; (F) PPP activation as judged by relative levels of labeled 6P-gluconate to hexose phosphate. (G-H) CS testing of mice (n = 12) showed that G6PD Med− mice maintained a significantly faster CS (8% increase). Dashed lines indicate CS. (I) G6PD Med− mice have higher anaerobic work capacity (AWC) as measured by the slope, hG6PD ND = 1715 and hG6PD MED- = 945.4 (significance, ∗ P < .05, ∗∗ P < .01, ∗∗∗∗ P < .0001).

    Journal: Blood Advances

    Article Title: Increased exercise tolerance in humanized G6PD-deficient mice

    doi: 10.1182/bloodadvances.2024013968

    Figure Lengend Snippet: Characterization of G6PD Med− mice. (A) mQTL analysis of samples from 13 091 blood donors highlights correlation of succinate levels with polymorphic G6PD. (B) Locus zoom identifying G6PD as a target of interest. (C) An overview of glucose metabolism in redox homeostasis in RBCs. (D) Total abundance of 6-phosphogluconate (6P-gluconate) from metabolic tracing (sum of labeled and unlabeled) present in genotypes. (E) G6PD activity assay; (F) PPP activation as judged by relative levels of labeled 6P-gluconate to hexose phosphate. (G-H) CS testing of mice (n = 12) showed that G6PD Med− mice maintained a significantly faster CS (8% increase). Dashed lines indicate CS. (I) G6PD Med− mice have higher anaerobic work capacity (AWC) as measured by the slope, hG6PD ND = 1715 and hG6PD MED- = 945.4 (significance, ∗ P < .05, ∗∗ P < .01, ∗∗∗∗ P < .0001).

    Article Snippet: G6PD activity was quantitated using the G6PD reagent set (Pointe Scientific, catalog no. G7583180) following the manufacturer’s protocol, unless noted otherwise.

    Techniques: Labeling, Activity Assay, Activation Assay